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Biblioteca (s) : |
INIA La Estanzuela. |
Fecha : |
23/03/2018 |
Actualizado : |
09/10/2019 |
Tipo de producción científica : |
Artículos en Revistas Indexadas Internacionales |
Autor : |
OLINDA, R. G.; MAIA, L. A.; FRADE, M. T. S.; SOARES, M. P.; BARROS, S. S.; DRIEMEIER, D.; RIET-CORREA, F.; DANTAS, A. F. M. |
Afiliación : |
Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG); Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG); Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG); Laboratório Regional de Diagnóstico, Faculdade de Veterinária (FV), Universidade Federal de Pelotas (UFPel); Laboratório Regional de Diagnóstico, Faculdade de Veterinária (FV), Universidade Federal de Pelotas (UFPel); Departamento de Patologia Clínica Veterinária, Faculdade de Veteriná- ria, Universidade Federal do Rio Grande do Sul (UFRGS),; FRANKLIN RIET-CORREA AMARAL, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG); Programa de Pós-graduação em Medicina Veterinária, Hospital Veterinário, Centro de Saúde e Tecnologia Rural, Universidade Federal de Campina Grande (UFCG). |
Título : |
Degenerative axonopathy associated with copper deficiency in pigs. (Axonopatia degenerativa associada com deficiência de cobre em suínos). |
Fecha de publicación : |
2017 |
Fuente / Imprenta : |
Pesquisa Veterinária Brasileira, Rio de Janeiro v. 37, n. 9, p. 911-915, setembro. 2017. |
DOI : |
10.1590/S0100-736X2017000900002 |
Idioma : |
Inglés |
Notas : |
Article history: 1 Received on May 29, 2016 // Accepted for publication on November 11, 2016. |
Contenido : |
Abstract: The epidemiological, clinic and morphological (pathological and ultrastructural) aspects of four outbreaks of copper deficiency affecting 21- to 90-day-old pigs in the Northeast region of Brazil are reported. Clinical signs began with paraparesis and ataxia and progressed to flaccid or spastic paralysis of the pelvic and thoracic limbs, followed by sternal and/or lateral recumbence. In addition, some animals showed dog-sitting position and intention tremors. The clinical manifestation period was 5-20 days. Significant gross lesions were not observed; however, microscopically, symmetrical degeneration of the white matter with ballooned myelin sheaths containing occasional macrophages was observed, mainly in the spinal cord. Two pigs presented with necrosis ad loss of Purkinje cells and ectopic Purkinje cells in the granular layer and cerebellar white matter. A ultrastructural analysis showed different degrees of damage of myelinated axons in the spinal segments, including an absence of the axoplasm structures with only axonal residues remaining. The myelin sheaths were degenerated and often collapsed into the space previously occupied by the axon. These results suggest that myelin degeneration is secondary to the axonal lesion. Finally, the concentration of copper in the liver was determined using atomic absorption spectrophotometry and was found to be low (ranging from 2.2 to 10.8 ppm). In conclusion, in the Brazilian semiarid region, Cu deficiency occurs in 21 to 90-day-old pigs that ingested different types of waste in their food. MenosAbstract: The epidemiological, clinic and morphological (pathological and ultrastructural) aspects of four outbreaks of copper deficiency affecting 21- to 90-day-old pigs in the Northeast region of Brazil are reported. Clinical signs began with paraparesis and ataxia and progressed to flaccid or spastic paralysis of the pelvic and thoracic limbs, followed by sternal and/or lateral recumbence. In addition, some animals showed dog-sitting position and intention tremors. The clinical manifestation period was 5-20 days. Significant gross lesions were not observed; however, microscopically, symmetrical degeneration of the white matter with ballooned myelin sheaths containing occasional macrophages was observed, mainly in the spinal cord. Two pigs presented with necrosis ad loss of Purkinje cells and ectopic Purkinje cells in the granular layer and cerebellar white matter. A ultrastructural analysis showed different degrees of damage of myelinated axons in the spinal segments, including an absence of the axoplasm structures with only axonal residues remaining. The myelin sheaths were degenerated and often collapsed into the space previously occupied by the axon. These results suggest that myelin degeneration is secondary to the axonal lesion. Finally, the concentration of copper in the liver was determined using atomic absorption spectrophotometry and was found to be low (ranging from 2.2 to 10.8 ppm). In conclusion, in the Brazilian semiarid region, Cu deficiency occurs in 21 to ... Presentar Todo |
Palabras claves : |
Axonal degeneration; COPPER DEFICIENCY; DEFICIENCIA DE COBRE; Degeneração axonal; Degeneração da mielina; Degenerative myelopathy; Mielopatia; MINERAL DEFICIENCY; Myelin degeneration; PLATAFORMA SALUD ANIMAL; SWINE. |
Thesagro : |
DEFICIENCIAS DE MINERALES; ENFERMEDADES DE LOS ANIMALES; SUINOS. |
Asunto categoría : |
E16 Enfermedades de los animales |
URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/8985/1/Pesq.-Vet.-Bras.-379-911-915-setembro-2017-Olinda.pdf
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Marc : |
LEADER 02918naa a2200397 a 4500 001 1058315 005 2019-10-09 008 2017 bl uuuu u00u1 u #d 024 7 $a10.1590/S0100-736X2017000900002$2DOI 100 1 $aOLINDA, R. G. 245 $aDegenerative axonopathy associated with copper deficiency in pigs. (Axonopatia degenerativa associada com deficiência de cobre em suínos).$h[electronic resource] 260 $c2017 500 $aArticle history: 1 Received on May 29, 2016 // Accepted for publication on November 11, 2016. 520 $aAbstract: The epidemiological, clinic and morphological (pathological and ultrastructural) aspects of four outbreaks of copper deficiency affecting 21- to 90-day-old pigs in the Northeast region of Brazil are reported. Clinical signs began with paraparesis and ataxia and progressed to flaccid or spastic paralysis of the pelvic and thoracic limbs, followed by sternal and/or lateral recumbence. In addition, some animals showed dog-sitting position and intention tremors. The clinical manifestation period was 5-20 days. Significant gross lesions were not observed; however, microscopically, symmetrical degeneration of the white matter with ballooned myelin sheaths containing occasional macrophages was observed, mainly in the spinal cord. Two pigs presented with necrosis ad loss of Purkinje cells and ectopic Purkinje cells in the granular layer and cerebellar white matter. A ultrastructural analysis showed different degrees of damage of myelinated axons in the spinal segments, including an absence of the axoplasm structures with only axonal residues remaining. The myelin sheaths were degenerated and often collapsed into the space previously occupied by the axon. These results suggest that myelin degeneration is secondary to the axonal lesion. Finally, the concentration of copper in the liver was determined using atomic absorption spectrophotometry and was found to be low (ranging from 2.2 to 10.8 ppm). In conclusion, in the Brazilian semiarid region, Cu deficiency occurs in 21 to 90-day-old pigs that ingested different types of waste in their food. 650 $aDEFICIENCIAS DE MINERALES 650 $aENFERMEDADES DE LOS ANIMALES 650 $aSUINOS 653 $aAxonal degeneration 653 $aCOPPER DEFICIENCY 653 $aDEFICIENCIA DE COBRE 653 $aDegeneração axonal 653 $aDegeneração da mielina 653 $aDegenerative myelopathy 653 $aMielopatia 653 $aMINERAL DEFICIENCY 653 $aMyelin degeneration 653 $aPLATAFORMA SALUD ANIMAL 653 $aSWINE 700 1 $aMAIA, L. A. 700 1 $aFRADE, M. T. S. 700 1 $aSOARES, M. P. 700 1 $aBARROS, S. S. 700 1 $aDRIEMEIER, D. 700 1 $aRIET-CORREA, F. 700 1 $aDANTAS, A. F. M. 773 $tPesquisa Veterinária Brasileira, Rio de Janeiro$gv. 37, n. 9, p. 911-915, setembro. 2017.
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Registro original : |
INIA La Estanzuela (LE) |
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Registro completo
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Biblioteca (s) : |
INIA Las Brujas; INIA Tacuarembó. |
Fecha actual : |
21/02/2014 |
Actualizado : |
19/11/2019 |
Tipo de producción científica : |
Artículos en Revistas Agropecuarias |
Autor : |
MONTOSSI, F.; DE BARBIERI, I.; CIAPPESONI, G.; RAVAGNOLO, O.; MEDEROS, A.; SOARES DE LIMA, J.M.; DE MATTOS, D.; PÉREZ JONES, J.; FROS, J.; GRATTAROLA, M. |
Afiliación : |
FABIO MARCELO MONTOSSI PORCHILE, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; LUIS IGNACIO DE BARBIERI ETCHEBERRY, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; CARLOS GABRIEL CIAPPESONI SCARONE, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; OLGA RAVAGNOLO GUMILA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; AMERICA ESTHER MEDEROS SILVEIRA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; JUAN MANUEL SOARES DE LIMA LAPETINA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; DANIEL DE MATTOS, Carne Hereford del Uruguay S.A.; J. PÉREZ JONES, SCMAU (Sociedad de Criadores Merino Autraliano del Uruguay); J. FROS, SCMAU (Sociedad de Criadores Merino Autraliano del Uruguay); M. GRATTAROLA, SUL (Secretariado Uruguayo de la Lana). |
Título : |
Merino fino: una experiencia innovadora de mejoramiento genético |
Fecha de publicación : |
2005 |
Fuente / Imprenta : |
Revista INIA, 2005, no. 5, p. 5-8 |
ISSN : |
1510-9011 |
Idioma : |
Español |
Contenido : |
A partir del año 1998, el INIA, el SUL y la Sociedad de Criadores Merino Australiano del Uruguay (SCMAU) coordinaron esfuerzos para la ejecución conjunta de un Proyecto de Investigación y Desarrollo del Merino Fino para Uruguay, con el objetivo de desarrollar una alternativa de producción ovina para mejorar la sustentabilidad socioeconómica de los productores de lana de las regiones de Basalto y otras regiones de problemática similar. |
Palabras claves : |
LANAS FINAS; NMF (NÚCLEO FUNDACIONAL DE MERINO FINO). |
Thesagro : |
MEJORAMIENTO ANIMAL; MEJORAMIENTO GENETICO ANIMAL; MERINO; OVINOS; RAZAS (ANIMALES). |
Asunto categoría : |
-- L01 Ganadería |
URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/6822/1/re-5-Montossi-F.-p.5-8.pdf
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Marc : |
LEADER 01313naa a2200325 a 4500 001 1012566 005 2019-11-19 008 2005 bl uuuu u00u1 u #d 022 $a1510-9011 100 1 $aMONTOSSI, F. 245 $aMerino fino$buna experiencia innovadora de mejoramiento genético 260 $c2005 520 $aA partir del año 1998, el INIA, el SUL y la Sociedad de Criadores Merino Australiano del Uruguay (SCMAU) coordinaron esfuerzos para la ejecución conjunta de un Proyecto de Investigación y Desarrollo del Merino Fino para Uruguay, con el objetivo de desarrollar una alternativa de producción ovina para mejorar la sustentabilidad socioeconómica de los productores de lana de las regiones de Basalto y otras regiones de problemática similar. 650 $aMEJORAMIENTO ANIMAL 650 $aMEJORAMIENTO GENETICO ANIMAL 650 $aMERINO 650 $aOVINOS 650 $aRAZAS (ANIMALES) 653 $aLANAS FINAS 653 $aNMF (NÚCLEO FUNDACIONAL DE MERINO FINO) 700 1 $aDE BARBIERI, I. 700 1 $aCIAPPESONI, G. 700 1 $aRAVAGNOLO, O. 700 1 $aMEDEROS, A. 700 1 $aSOARES DE LIMA, J.M. 700 1 $aDE MATTOS, D. 700 1 $aPÉREZ JONES, J. 700 1 $aFROS, J. 700 1 $aGRATTAROLA, M. 773 $tRevista INIA, 2005, no. 5, p. 5-8
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